The
Cavernous Venous Sinus |
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This is clinically very important on account of the following:
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Location | |
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Relations | |
Medially: Pituitary gland(1) and sphenoidal air cells(2). Superiorly: Temporal lobe of the brain(3). Laterally: Trigerminal ganglion and nerve. Anteriorly: The contents of the superior orbital fissure and ................the optic nerve. Posteriorly: Petrous temporal bone. Within: Lateral wall - Oculomotor, trochlear, ophthalmic and ...........maxillary nerves. In it: Carotid siphon with its associated sympathetic plexus, ........the abducens nerve and venous blood. |
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Inter-cranial communications | |
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Extracranial communications | |
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Clinical correlates on the cavernous venous sinus | |
Spread of infections | |
Danger area of the face Infections from here spread to cavernous sinus via opthalmic veins
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Cavernous sinus thrombosis |
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A common effect of infection of in a venous sinus is thrombosis. The risk is higher in the cavernous sinus due to the slow movement of blood. The effects are usually:
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Arteriovenous fistula | |
This may be produced by fractures of the bases of the skull, in which the internal carotid artery tears within the cavernous sinus. Arterial blood rushes in to the sinus, enlarging it and forcing blood out of it through the communicating veins. This causes exophthalmos and chemosis on the side of the injury. In these circumstances, the bulging eye pulses in synchrony with the radial (or any other) pulse. This condition is called pulsatile exophthalmos. |
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Others |
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Infections spreading to the sinuses may affect the pituitary and the many cavernous sinus communications may provide alternative routes for spread of malignancies. | |